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Fattening the Fat Cells With a Drug to Live Longer?

Ask the New York Times

Majid Ali, M.D.


On November 3, 2011, The New York Times ran a front-page story entitled "Prospect of Delaying Aging Ills Is Raised in Cells Study of Mice." The article carried two remarkable pictures: (1) a fat mouse sitting next to a thin mouse; and (2) bloated fat cells (of the fat mouse) juxtaposed to normal-sized fat cells of the thin mouse. (

 

Fattened Unhealthy Fat Cells of a Drugged  Mouse (Left) Compared With the Normal Healthy Fat Cells of an Un-drugged Mouse (Right)

In the experiment, the removal of the senescent cells caused bloating of the normal fat cells.

So, the idea is to fatten the fat cells with a drug to extend the life span. Remarkable, isn’t it?

Good science is good science. In basic sciences, the important discoveries often lead to clinical benefits years or decades later. So, I welcome information about how the genetic pathways of the P16 gene and its related genes—the target of future drugs in the new report—can be altered to prolong life. What surprised me was that the Times completely ignored the obvious problem in its article: All available scientific data worldwide demonstrate that fattened fat cells produce more fattening hormones and obesity increases the risk of all inflammatory and degenerative disorders.

Fattening the Fat Cats

If I were a betting man, I would put a 500-to-one dollar wager against the Times prediction helping any obese people, except those who will rake hundreds of millions of dollars from the Wall Street buzz created by the Times. This, of course, is not the first time the Times would fatten the financial fat cats by its journalistic prowess. Consider how it fattened the fat cats by its reportage of the Leptin fiasco in the following quotes:


The Times Leptin Story August 1, 1995

The discovery of a fat-signaling hormone last week has given new insight into the body's system of fat control and suggested promising new practical approaches to the treatment of obesity. It may help solve the puzzles of how the body knows how much fat it has and why a person's weight seems always to be forced back to the set point.

The Times Leptin Story June 15, 1998

A potential anti-obesity drug being developed by Amgen Inc., which made headlines a few years ago by sharply reducing body fat in mice, has now been found to contribute to weight loss in people as well, scientists reported yesterday

The Times Leptin Story Jul 27, 1995

... speculators started buying Amgen's stock, raising its price by $4.375...


The Times’s Lapdog Joes and Its Untold Leptin-Insulin-Toxicity Story

Altering leptin-associated genetic pathways also causes liver injury (hepatic steatosis), insulin toxicity, Type 2 diabetes, and related metabolic disorders. No, the Times never considered it appropriate to apologize to its readers for hyping the “leptin drug” and fattening the fat cats at the expense of the gullible. The Times has never done a comprehensive review of insulin toxicity —the most important root cause of weight gain, obesity, and Type 2 diabetes—and show how proper diet, spices, herbs, and nutrients can reduce insulin toxicity.

The Drugged Mice Really Didn't Live Longer

On page four, the Times noted that the drugged mice in the study had not lived longer than the un-drugged mice. So, the bit about mice living longer was a hope, not something really observed. This sort of reporting in not uncommon among lapdog Joes at The Times—journalists who once aspired to be watch dogs but were turned into lapdogs by their corporate paymasters. They promote what their paymasters demand from them. They were once idealists with high ethical standards before they became tired, dispirited, and lost all sense of ethics. (Go to www.lapdogjoes.com for my several articles on lapdog Joes of the Times and The New Enron Journal of Medicine.)

The Times has published a large number of articles to promise its readers extended life span with drugs. The paper has not been interested in reviewing the results of its drug promotion years after spread the word. So its readers are generally not aware how they did after taking the promised life-extending drugs or how toxic they became with the drugs. I address this problem in my book Oxygen and Aging (2000) and summarized it in the article on aging on Wiki-medical encyclopedia entitled “Oxygen Governs the Aging Process.”

What I lament in this context is that not even one cent of taxpayers dollars allocated for gene research are allowed to go to research on the uses of oxystatic therapies to promote health, prevent disease, and so extend life. The Times is peculiarly and consistently silent on this critical issue, while it runs one after the other gene strories on its front pages.

Fattening the Fat Cells to Extend Life Fattened By a Drug

The picture of two 9-month-old mice given below is taken from the Times article (originally from the research paper). The one on the right received the drug to eliminate senescent cells. research paper

 

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