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More Direct Evidence for Oxygen Model of Diabetes
Majid Ali, M.D.



On April 14, 2014, the journal PLOS One published additional direct evidence for my Oxygen Model of Diabetes. The study was conducted to investigate the effects of hypoxia (lack of oxygen) at high altitudes on Mount Everest climbers. Its core is that finding is that hypoxia increased blood levels of markers of inflammation and oxidative stress known to lead to insulin resistance. Before continuing with the results of the study, below is a brief description of Oxygen Model of Diabetes.

 

What Do Dead Starfish and Dead Coffee Plants Teach About Diabetes Pandemic?

 

 

Oxygen Model of Diabetes

My Oxygen Model of Diabetes is a unifying model that explains all aspects of diabetes Types 1 and 2—causes, clinical course, consequences, and control—on the basis of impaired oxygen signaling, diminished oxygen’s detergent functions, and interrupted oxygen’s cellular detox and repair functions. I put forth this model first in a series of articles in 2000 and presented it at length it in my book “Dr. Ali’s Plan for Reversing Diabetes (2006)” (available for download at www.aliacademy.org)

In my book, I illustrated the insulin/insulin receptor dysfunction with a crank/crank-shaft analogy.Briefly, the cell membranes become resistant to insulin when they become chemicalized—plasticized, so to speak—and hardened, immobilizing the insulin receptors embedded in the membranes. The insulin receptor is a protein that criss-crosses the cell membrane like a cord. One of the consequences of grease buildup on cell membranes is that insulin receptor becomes turned and twisted, literally and figuratively. In a previous paper, I offered the analogy of a crank and a crank-shaft to explain insulin resistance. I visualize insulin as a crank—a device that transmits rotary motion—and the insulin receptor protein as a crank-shaft embedded in the cell membrane.

During the Everest expedition in 2007, 24 people traveled to the mountain and were checked for blood sugar control, body weight changes and signs of inflammation at base camp, which was at an altitude of 5,300 meters (about 17,388 feet).

Half of the participants remained at base camp while the other half climbed Everest to a maximum altitude of 8,848 meters (29,028 feet). Measurements were taken in each group at week six and week eight of the trek.

"These results have given us useful insight into the clinical problem of insulin resistance. Fat tissue in obese people is believed to exist in a chronic state of mild hypoxia because the small blood vessels are unable to supply sufficient oxygen to fat tissue," study leader Mike Grocott, a professor of anesthesia and critical care at the University of Southampton, said in a university news release.

"These results have given us useful insight into the clinical problem of insulin resistance. Fat tissue in obese people is believed to exist in a chronic state of mild hypoxia because the small blood vessels are unable to supply sufficient oxygen to fat tissue," study leader Mike Grocott, a professor of anesthesia and critical care at the University of Southampton, said in a university news release.

Scientists were able to observe things in healthy people at high altitudes that normally are only seen in obese people at sea level, Grocott noted.

 

k  Dr. Ali's Diabetes Course

k Diabetes Pandemic

k Subtypes of Diabetes Type 2

k  The Oxyegn Model of Diabetes

k  Evidence for Oxygen Model of Diabetes - Mt. Everest Study

k The Oxygen Model of Obesity

k How Blood Cells Tell the Diabetes Story?

k What Does a Red Blood Cell Tell - About Diabetes

k What Do Blood Platelet Tell About Diabetes

k What Do Blood Corpuscles Tell About Diabetes -The Taurine Story

k Blood Pictures Tell the Blood Story - AA Oxidopathy

k Dr. Ali's Course on Oxygen

k  Oxygen Literacy

k  Oxygen Intelligence

k  Oxygen Is the Primal Nutrient

k  History of Oxygen

k  Oxygen's Three-Legged Throne

 

k Insulin Evolutionary

k Seven Stages of Insulin Toxicity

k Less Insulin, More Life

k Evidence for Insulin Toxicity

k  Insulin Ranges Data

k If Mice Can Reverse Diabetes, Why Can't People?

k Dysox Explains the Exercise-Weight-Loss Disconnect

 

* Hypoglycemia: Diagnosis and Treatment
Insulin Toxicity
Weight and Obesity
Diabetes
Tutorial GG.16 The Dysox Model of Diabetes and De-Diabetization Potential. Townsend Letter-The examiner of Alternative Medicine. 2007; 286:137-145.

* Tutorial GG.31 Ali M. Oxygen, Insulin Toxicity, Inflammation, And the Clinical Benefits of Chelation. Part I. Townsend Letter-The examiner of Alternative Medicine. 2009;315:105-109. October, 2009.

* Tutorial GG.32 Ali M. Insulin Reduction and EDTA Chelation: Two Potent and Complementary Approaches For Preventing and Reversing Coronary Disease. Oxygen, Insulin Toxicity, Inflammation, and the Clinical Benefits of Chelation - Part II. Townsend Letter-The examiner

 

 

 

 

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