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Dengue Fever

Part Two: Old Empirical and Newer Nondrug Therapies

Majid Ali, M.D.

Please read Part One of this tutorial for an overview of the global perspective on dengue fever. Both parts of this tutorial are presented in video format at YouTube.Enter "Majid Ali, M.D." at www.youtube.com for full directory of my library.

The Signs and Symptoms of Dengue Infection

The signs and symptoms of dengue infection include fever, headache, a measles-like rash, and muscle pain. The severity of bone pain is often marked and accounts for its "breakbone" designation. The primary mechanism underlying the disabling myalgia and bone pain is oxidative coagulopathy triggered by the virus-related acidity (acidosis), incremental oxyradical activity (oxidosis), and clotting-unclotting dysequilibrium (CUD). I urge readers to view my You Tube seminar entitled "The Sour Blood State" for a vocal narrative on these subjects. Acidosis, oxidosis, and CUD then lead to sludging of red blood corpuscles, clumping platelets, congealing of plasma, and the formation of micro-clots and micro-plaques in the circulating blood.

Dr. Aliís Guidelines for the Treatment of Dengue Fever

The following guidelines are based on extensive personal experience with acute viral infections which cause oxidative coagulopathy. All of the following items are discussed in YOU TUBE(videos. Please enter "Majid Ali, M.D." on Youtube and search specific videos pertaining to the subject.

* Optimal hydration (consider soups for the salt needs)

* Ginger/lemon tea

* Hydrogen Peroxide Foot Soaks / Three times a day

* Anti-inflammatory oil rubs over the abdomen, chest, and other painful areas

* Anti-inflammatory enzyme therapies to prevent blood and vascular complications

* Turmeric (one teaspoon) plus vitamin C one gram / Three times a day

* Multivitamin tablets / one tablet three times a day

* Intramsucular nutrient injections / daily for three days for severe infection

* Intravenous hydrogen peroxide therapy / daily for three days intravenous for severe infections

EDTA for Treating Dengue Fever

I published several research papers to report my observations concerning the clinical value of EDTA for arresting and reversing damage to blood plasma and corpuscles, as well as the endo cells lining the blood vessels caused by oxidative coagulopathy (see below for descriptions). Some experienced pharmacists who are familiar with my work report the beneficial effects of one tablet of EDTA (500 mg) taken three times a day.

Oxidative Coagulopathy

In 1997, my colleague, Omar Ali, and I described the following seven morphologic stages of oxidative coagulopathy observed with high-resolution phase-contrast microscopy of freshly-prepared and unstained peripheral blood smears:

1. Erythrocyte and Leukocyte Membrane Deformities

2. Diaphanous Congealing of Plasma

3. Platelet Aggregation and Membrane Damage

4. Filamentous Coagulum (Fibrin Needles)

5. Lumpy Coagulum

6. Microclots

7. Microplaques

For illustrative photomicrographs and for reading more on the subject, please click Oxidative Coagulopathy.

Membrane Damage of Red Blood Cell (Erythrocytes) and White Blood Cells (Leukocytes)

Red blood cells when observed with an ordinary bright-light microscope appear as rigid biconcave disc-shaped corpuscles. When examined with high-resolution, phase-contrast microscope, these cells are seen as malleable round cells that readily change their shape to ovoid, triangular, dumb-bell or irregular outlines to squeeze past other erythrocyte in densely populated fields. Such cells assume their regular rounded contour as soon as they find open space. The earliest and most common abnormality we observed in oxidative coagulopathy was erythrocytic rouleaux formation, erythrocyte lysis, leucocytic clumping, and platelet aggregation. Notably, we often observed erythrocytic and leucocytic changes of coagulopathy independent of platelet clumping. Frequently observed erythrocytic membrane abnormalities included wrinkling, tear drop deformity, sharp angulations and spike formations. We established the oxidative nature of such abnormalities by demonstrating their reversibility with antioxidants such as taurine, vitamin E, vitamin A, and vitamin C reported previously but not shown in this article. Parenthetically, we add that we have observed similar evidence of erythrocyte membrane injury in diverse clinical entities associated with accelerated molecular injury such as disabling chronic fatigue, fibromylagia and a host of severe nutritional, ecologic and autoimmune disorders

The Pathogenic Mechanisms of Endothelial Injury

The pathogenic mechanisms of the severe forms of the disease (Dengue shock syndrome (DSS) and Dengue hemorrhagic fever (DHF) involve endothelial damage. A large number of molecular pathways for such injury have been proposed, including: (1) impaired coagulation pathways; (2) abnormal inflammatory pathways; (3) increased levels of soluble thrombomodulin (sTM); (4) formation of abnormal protein complexes (for instance, thrombomodulin-thrombin-protein C complex) at the endothelial surface; (5) reduction in activated protein C (APC); (6) by the inactivation of procoagulant factors Va and VIIIa); (7) abnormal responses of the endothelial protein C receptor; (8) alterations in the antithrombotic and cytoprotective protein C pathways; and (9) altered expressions of cellular adhesion molecules, such as selectins.

I point out that all of the above factors play secondary roles to primary insults of acidosis, oxidosis, and clotting-unclotting dysequilibrium, which we proposed as the primary insults to all forms of endothelial injury in oxidative coagulopathy.

Related Tutorials

* Infectious Diseases

* Viruses and Virology

* Dengue Fever Part One

* Dengue Fever Part Two

* Introduction to Inflammation and Immunity

* What Is Inflammation?

* Infectious Diseases

* Castor Oil Rubs for Colicky Babies and Children

Sesame Oil: Why Is It One of My Darlings?

*  Anti-Inflammatory Spice Therapies for Arthritis

Anti-Inflammatory Topical Oil Therapies for Arthritis

* LAPs and TAPs Determine the Outcome in Infections Everywhere in the Body

 

 

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