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A-Fib (Atrial Fibrillation), Stroke, and Pacemaker Ablation
Majid Ali, M.D.
 


Here are the main points of this article

* A-Fib is a form of irregular heartbeat.
* About three million Americans have atrial fibrillation (A-Fib).
* A-Fib is caused multiple irregular electrical waves created by a heart pacemaker under stress.
* In A-Fib, atria, the heartís upper chambers, contract in an coordinated, irregularl, and rapid way, sending uncoordinated erratic signals to the ventricles (the lower chambers) that pump blood into the rest of the body.
* A-Fib is caused by dysfunctional oxygen signaling in the cells of the heartís pacemaker.
* Dysfunctional oxygen signaling (dysox) which sets the stage for A-Fib is usually associated with rapid heart rate, bouts of palpitations, and excess adrenaline.
* Both cardiac myocytic (muscle) disease and coronary artery disease increase the risk of A-Fib. The root cause of both is is also dysfunctional oxygen signaling.
* A-Fib carries a higher risk of stroke.
* A-Fib also carries a higher risk of changing into more serious forms of cardiac rhythm disorders.
*  The prevention and control is best achieved by safely addressing all threats to oxygen signaling in any part of the body by natural methods and under the close supervision of a cardiologist.
* A-Fib initially requires treatment with drugs that slow the heart rate.
* A-Fib is treated with procedure called radio-frequency ablation when drug therapies fail.

Heart Pacemaker Destruction (Radiofrequency Ablation)

The decision to have or defer the destruction of the heart pacemaker with radio-frequency requires a careful consideration of all aspects of an individual case. To assist those learning about natural measures for slowing the heart rate and preventing complications of A-Fib, I offer a video seminar on tghe subject, which can be downloaded from www.aliacademy.org.
 

List of Related Articles

* A-Fib (Atrial Fibrillation), Stroke, and Pacemaker Ablation

*  Atrial Fibrillation: When the Heart Rebels Against the Mind

Seeing the Heart

* Coronary Heart Disease - Simplified

* Being Oneís Own Cardiologist

* Anatomy of the Heart

* Physiology of the Heart

* Saving a Rushed Heart From Itself

* Cardiac Myocytic Disease (CMD) 

* What Causes Coronary Heart Disease?

* Pediatric Cardiac Dysfunction State

* A-Fib (Atrial Fibrillation), Stroke, and Pacemaker Ablation

*  Atrial Fibrillation: When the Heart Rebels Against the Mind

* Blood Clots and Unclots At All Times

* More Coronary Plaques, Fewer Heart Deaths

* The More-Plaques-Fewer-Deaths Paradox,  Part B

* Should You Say No To Non-emergency Coronary Stent?

* What Do Cholesterol Crystals in Arterial Walls Mean?

* HDL Cholesterol Is Dignified Cholesterol

 

Tutorial He8  The More-Plaques-Fewer-Deaths Paradox  Part II

Tutorial He9  The More-Plaques-Fewer-Deaths Paradox  Part III

Tutorial He10  The More-Plaques-Fewer-Deaths Paradox  Part IV

Tutorial He11  The More-Plaques-Fewer-Deaths Paradox  Part V 

Tutorial GG12. The dysox model of coronay artery disease. Townsend Letter for Doctors and Patients. 2006;270/71:110-112.

Healing Stories

*A Bullet for Hypertension
*Conversations With *Angels
*The Bite of the Neck Muscles
*The Sword Story
*The Bite of the Grey Dog
* I Don't want Dialysis

* Cardiac Cellular Disease (CCD)

* Tutorial GG.12 Ali M. Fischer S, Juco J, et al. The dysox model of coronay artery disease. Townsend Letter for Doctors and Patients. 2006;270/71:110-112.

Tutorial GG.7 Ali M. Oxygen governs the inflammatory response and adjudicates the man-microbe conflicts. Townsend Letter for Doctors and Patients. 2005;262:98-103.

Tutorial GG.31 Ali M. Oxygen, Insulin Toxicity, Inflammation, And the Clinical Benefits of Chelation. Part I. Townsend Letter-The examiner of Alternative Medicine. 2009;315:105-109. October, 2009.


END
 

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