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Hormone Replacement Therapy (HRT) or

Receptor Restoration Therapy (RRT)?

Majid Ali, M.D.


Some recent review articles addressed the issue of hormone replacement therapy (HRT) for menopausal women and concluded that such therapy is appropriate for most women. The primary argument is that the potential for such therapy for prevention of coronary artery disease (CAD) and osteoporosis far outweighs its risk of breast, uterine and other cancers. This article challenges that opinion and puts forth a counterview of "oxidative menopausal dysfunction " (OMD-II) that is based on microecologic cellular and macroecologic tissue-organ disruptions of the body that lead to CAD, osteoporosis, and other menopausal symptomatology. For addressing health issues of postmenopausal women, OMD-II approach shifts the focus from the use of carcinogenic synthetic hormones to restoration of human ecosystems by grounded its therapies on the established knowledge of redox, acid-base, and enzymatic homeostasis.

From the Departments of medicine of capital University of Integrative Medicine, D.C., and Institute of Integrative Medicine, Denville, N.J., and New York, and the Department of Pathology of the College of Surgeons and Physicians of Columbia University, New York.

This article addresses the following seven issues: (1) the roles of oxidative disruptions of the bowel, blood, and liver ecosystems in the pathophysiology of the "menopausal syndrome"; (2) empirical observations concerning control of symptom-complexes of the menopausal syndrome with therapies directed at ecologic restorations without the use of synthetic hormones; (3) comparative efficiencies of OMD-II and HRT approaches to prevention of CAD; (4) the value of those two approaches to prevention of osteoporosis; (5) the pros and cons of those approaches to problems of vasomotor symptoms of menopausal syndrome and risk of thromboembolism; and (7) the merits of those two very different strategies to the global goal of improving the general health, quality of life, and life expectancy. Arguments are marshalled for the view that the optimal management of peri- and postmenopausal syndromes requires that the primary focus be on addressing all sources of accelerated oxidative stress on microecologic cellular and marcoecologic tissue-organ systems of the body. Furthermore, restoration of such ecosystems must be undertaken before resorting to the use of synthetic hormones for control of menopausal symptoms.


(Abstract of an article first published in 1998)

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